Download Adverse Cutaneous Drug Reactions to Cardiovascular Drugs by Esen Özkaya, Kurtuluş Didem Yazganoğlu PDF

By Esen Özkaya, Kurtuluş Didem Yazganoğlu

Adverse cutaneous drug reactions (ACDR) are one of the such a lot common occasions in sufferers receiving drug remedy. Cardiovascular (CV) medicines are a huge staff of substances with capability chance of constructing ACDR particularly in aged as advertising of extra new medicines and their prescription proceed to extend. even if, like with such a lot different medicinal drugs the precise occurrence of cutaneous uncomfortable side effects from CV medications is hard to estimate because of sporadic reporting. additionally, a competent designation of a undeniable drug because the reason behind a undeniable form of response can hardly ever be made. except the well known angioedema/urticaria from ACE inhibitors, lichen planus / lichenoid response from beta adrenergic blockers and photosensitivity from thiazid diuretics, ACDR from CV medications could be obvious in a large spectrum extending to infrequent yet life-threatening stipulations corresponding to erythroderma, Stevens-Johnson syndrome, poisonous epidermal necrolysis or drug hypersensitive reaction syndrome. during this accomplished evaluate, the mentioned different types of ACDR to CV medications could be mentioned in response to drug classification and the kind of dermatologic response with particular emphasize on cross-reactions and the function of patch trying out in diagnosis.

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29). The lips (Figs. 31), hands, and genitalia (especially male genitalia) are frequently involved sites. FDE on the lips might be difficult to differentiate from herpes simplex infection. Mucosal lesions are usually bullous/ erosive (Fig. 32) but they may also present with aphthous or erythematous morphology [20]. Bullous/erosive mucosal lesions might be difficult to differentiate from pemphigus vulgaris, EM major, or SJS [20]. Aphthous mucosal lesions should be differentiated from aphthous stomatitis, herpes simplex infection, and Behçet’s disease.

Unlike the true serum sickness, a type III hypersensitivity reaction, however, vasculitis and fever are not consistent findings in the reported cases of SSLR. Besides, gastrointestinal disturbances, lymphadenopathy, low complement levels, circulating immune complexes, and proteinuria associated with renal involvement are usually absent or rarely reported in drug-induced SSLR [4, 73]. ” Penicillins, cephalosporins, sulfonamides, and many other drugs have been implicated to cause drug-induced serum sickness or SSLR.

Cardiovascular drugs including statins have been reported among the main inducers of drug-induced pruritus [74]. Flushing may occur with vasodilators and CCBs. Xerosis is a common side effect of lipid-lowering drugs. Peripheral edema is frequently associated with the use of adrenergic neuron blockers (reserpine), CCBs, and vasodilators. Some drugs may cause ulceration of the skin and mucosa. There are increasing reports on perianal, peristomal, oral, or skin ulcerations with nicorandil, a vasodilatory drug used to treat angina [75].

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